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Listed below are some cancer-related journal articles:

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Suppression of pRB expression in normal human mammary epithelial cells is associated with resistance to all-trans-retinoic acid but not N-(4-hydroxylphenyl)-retinamide

Human Papillomavirus Type 16 E6 Inactivation of p53 in Normal Human Mammary Epithelial Cells Promotes Tamoxifen-mediated Apoptosis1

Inhibition of Retinoic Acid Receptor Function in Normal Human Mammary Epithelial Cells Results in Increased Cellular Proliferation and Inhibits the Formation of a Polarized Epithelium in Vitro

Tamoxifen but Not 4-Hydroxytamoxifen Initiates Apoptosis in p53(2) Normal Human Mammary Epithelial Cells by Inducing Mitochondrial Depolarization*

Retinoic Acid Receptor-beta2 Promoter Methylation in Random Periareolar Fine Needle Aspiration

Retinoids and Retinoic Acid Receptors Regulate Growth Arrest and Apoptosis in Human Mammary Epithelial Cells and Modulate Expression of CBP/p300

Dysregulated Expression of Cyclin D1 in Normal Human Mammary Epithelial Cells Inhibits All-trans-Retinoic Acid-Mediated G0/G1-Phase Arrest and Differentiation in Vitro

Interferon-regulatory factor-1 is critical for tamoxifen-mediated apoptosis in human mammary epithelial cells

Retinoic acid receptor β2 inhibition of metastasis in mouse mammary gland xenografts

Retinoic Acid-mediated G1-S-Phase Arrest of Normal Human Mammary Epithelial Cells Is Independent of the Level of p53 Protein Expression

Tamoxifen and tamoxifen ethyl bromide induce apoptosis in acutely damaged mammary epithelial cells through modulation of AKT activity